한국초파리연구자원은행 KDRC

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Papers citing KDRC – 번호, 제목, 조회, 등록일로 구성
No	Subject	Views	Date
16	Cellular and molecular basis of IR3535 perception in Drosophila	1651	22.05.26
15	Molecular and neuronal mechanisms for amino acid taste perception in the Drosophila labellum	1638	23.06.13
14	The insect somatostatin pathway gates vitellogenesis progression during reproductive maturation a...	1619	22.05.26
13	Histamine avoidance through three gustatory receptors in Drosophila melanogaster	1524	22.05.26
12	Drosophila melanogaster as potential alternative animal model for evaluating acute inhalation tox...	1388	24.05.13
11	Multisensory interactions regulate feeding behavior in Drosophila	1372	22.05.26
10	Tctp, a unique Ing5-binding partner, inhibits the chromatin binding of Enok in Drosophila	1341	23.05.16
9	Reduced branched-chain aminotransferase activity alleviates metabolic vulnerability caused by dim...	1314	23.05.16
8	Avoiding cantharidin through ionotropic receptors	1068	24.05.13
7	A Novel Interaction between MFN2/Marf and MARK4/PAR-1 Is Implicated in Synaptic Defects and Mitoc...	1066	24.05.13

Papers citing KDRC – 제목으로 구성
Subject
Cellular and molecular basis of IR3535 perception in Drosophila
Molecular and neuronal mechanisms for amino acid taste perception in the Drosophila labellum
The insect somatostatin pathway gates vitellogenesis progression during reproductive maturation and the post-mating response
Histamine avoidance through three gustatory receptors in Drosophila melanogaster
Drosophila melanogaster as potential alternative animal model for evaluating acute inhalation toxicity
Multisensory interactions regulate feeding behavior in Drosophila
Tctp, a unique Ing5-binding partner, inhibits the chromatin binding of Enok in Drosophila
Reduced branched-chain aminotransferase activity alleviates metabolic vulnerability caused by dim light exposure at night in Drosophila
Avoiding cantharidin through ionotropic receptors
A Novel Interaction between MFN2/Marf and MARK4/PAR-1 Is Implicated in Synaptic Defects and Mitochondrial Dysfunction

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