한국초파리연구자원은행 KDRC

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Papers citing KDRC – 번호, 제목, 조회, 등록일로 구성
No	Subject	Views	Date
16	Cellular and molecular basis of IR3535 perception in Drosophila	1811	22.05.26
15	Mitochondrial aconitase 1 regulates age-related memory impairment via autophagy/mitophagy-mediate...	1806	22.05.26
14	Drosophila melanogaster as potential alternative animal model for evaluating acute inhalation tox...	1792	24.05.13
13	The insect somatostatin pathway gates vitellogenesis progression during reproductive maturation a...	1771	22.05.26
12	Histamine avoidance through three gustatory receptors in Drosophila melanogaster	1631	22.05.26
11	History of Drosophila neurogenetic research in South Korea	1618	23.05.16
10	Reduced branched-chain aminotransferase activity alleviates metabolic vulnerability caused by dim...	1580	23.05.16
9	Tctp, a unique Ing5-binding partner, inhibits the chromatin binding of Enok in Drosophila	1514	23.05.16
8	Multisensory interactions regulate feeding behavior in Drosophila	1507	22.05.26
7	A Novel Interaction between MFN2/Marf and MARK4/PAR-1 Is Implicated in Synaptic Defects and Mitoc...	1449	24.05.13

Papers citing KDRC – 제목으로 구성
Subject
Cellular and molecular basis of IR3535 perception in Drosophila
Mitochondrial aconitase 1 regulates age-related memory impairment via autophagy/mitophagy-mediated neural plasticity in middle-aged flies
Drosophila melanogaster as potential alternative animal model for evaluating acute inhalation toxicity
The insect somatostatin pathway gates vitellogenesis progression during reproductive maturation and the post-mating response
Histamine avoidance through three gustatory receptors in Drosophila melanogaster
History of Drosophila neurogenetic research in South Korea
Reduced branched-chain aminotransferase activity alleviates metabolic vulnerability caused by dim light exposure at night in Drosophila
Tctp, a unique Ing5-binding partner, inhibits the chromatin binding of Enok in Drosophila
Multisensory interactions regulate feeding behavior in Drosophila
A Novel Interaction between MFN2/Marf and MARK4/PAR-1 Is Implicated in Synaptic Defects and Mitochondrial Dysfunction

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