한국초파리연구자원은행 KDRC

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Papers citing KDRC – 번호, 제목, 조회, 등록일로 구성
No	Subject	Views	Date
16	Drosophila melanogaster as potential alternative animal model for evaluating acute inhalation tox...	1897	24.05.13
15	Metabolic flux from the Krebs cycle to glutamate transmission tunes a neural brake on seizure onset	1892	22.05.26
14	Mitochondrial aconitase 1 regulates age-related memory impairment via autophagy/mitophagy-mediate...	1883	22.05.26
13	Cellular and molecular basis of IR3535 perception in Drosophila	1876	22.05.26
12	History of Drosophila neurogenetic research in South Korea	1660	23.05.16
11	Histamine avoidance through three gustatory receptors in Drosophila melanogaster	1656	22.05.26
10	Reduced branched-chain aminotransferase activity alleviates metabolic vulnerability caused by dim...	1632	23.05.16
9	Multisensory interactions regulate feeding behavior in Drosophila	1546	22.05.26
8	Tctp, a unique Ing5-binding partner, inhibits the chromatin binding of Enok in Drosophila	1541	23.05.16
7	A Novel Interaction between MFN2/Marf and MARK4/PAR-1 Is Implicated in Synaptic Defects and Mitoc...	1533	24.05.13

Papers citing KDRC – 제목으로 구성
Subject
Drosophila melanogaster as potential alternative animal model for evaluating acute inhalation toxicity
Metabolic flux from the Krebs cycle to glutamate transmission tunes a neural brake on seizure onset
Mitochondrial aconitase 1 regulates age-related memory impairment via autophagy/mitophagy-mediated neural plasticity in middle-aged flies
Cellular and molecular basis of IR3535 perception in Drosophila
History of Drosophila neurogenetic research in South Korea
Histamine avoidance through three gustatory receptors in Drosophila melanogaster
Reduced branched-chain aminotransferase activity alleviates metabolic vulnerability caused by dim light exposure at night in Drosophila
Multisensory interactions regulate feeding behavior in Drosophila
Tctp, a unique Ing5-binding partner, inhibits the chromatin binding of Enok in Drosophila
A Novel Interaction between MFN2/Marf and MARK4/PAR-1 Is Implicated in Synaptic Defects and Mitochondrial Dysfunction

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